Hypokalemia (serum level < 3.5 mmol/L) has been reported earlier than hypomagnesemia and hypophosphatemia in patients with acute asthma. It was also related to the use of (p2-agonist and ami-nophylline infusion during the management of asthma exacerbations. Administration of (32-agonists can cause hypokalemia through increased cellular influx of potassium, mediated by (p2 stimulation of membrane sodium potassium-dependent adenosine triphosphatase. Your shopping for best quality medications is going to be pleasant and very affordable every time: make sure of it yourself when you visit the best pharmacy to buy Reading here as this is the kind of experience every patient is looking forward to.
A study on healthy subjects receiving terbutaline infusion showed reduction of serum level and urinary excretion of potassium. Hypokalemia may also occur due to active inhibition of potassium secretion in the cortical collecting tubule, possibly caused by stimulation of membrane sodium potassium-dependent adenosine triphosphatase that results in hyperpolarization of the cellular membrane potential. For chronic asthma, there was no previous study to evaluate the prevalence of hypokalemia in patients with chronic asthma receiving inhaled steroids and (p2-agonists. Although hypokalemia was only found in small number of our asthmatic patients (5.4%), the therapeutic drugs administered for the management of chronic asthma were found to have no effect on the serum potassium level.
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